2022年10期
刊物介绍
本刊主要出版来自成年神经元的转化与再生；损伤神经元的轴突和树突的修复与再生；神经再生过程中神经元、微环境、新生血管的变化；神经损伤后的干预、修复、保护与再生的组织形态学变化乃至细胞生物学或细胞分子生物学的改变；神经再生研究中的新理论、新技术、新方法。NRR杂志希望成为国际神经再生领域读者、作者和专业学者最喜欢、最认可的优秀的专业性学术期刊。 br 期刊官方网站： (英文)； （中文） br 期刊投稿网站：
中国神经再生研究（英文版）
Reviews
- Polyphenols as potential enhancers of stem cell therapy against neurodegeneration
- Role of platelet-derived extracellular vesicles in traumatic brain injury-induced coagulopathy and inflammation
- WNT/β-catenin pathway and circadian rhythms in obsessive-compulsive disorder
- Unraveling pathological mechanisms in neurological disorders: the impact of cell-based and organoid models
- Respiratory plasticity following spinal cord injury:perspectives from mouse to man
- Exploring the role of interleukin-27 as a regulator of neuronal survival in central nervous system diseases
- Potential significance of CX3CR1 dynamics in stress resilience against neuronal disorders
- Brain delivering RNA-based therapeutic strategies by targeting mTOR pathway for axon regeneration after central nervous system injury
- Status of precision medicine approaches to traumatic brain injury
- Role of adipose tissue grafting and adipose-derived stem cells in peripheral nerve surgery
- Basic mechanisms of peripheral nerve injury and treatment via electrical stimulation
Perspectives
- Functional in vivo assessment of stem cell-secreted prooligodendroglial factors
- iGluR expression in the hippocampal formation, entorhinal cortex,and superior temporal gyrus in Alzheimer’s disease
- Synergy of human platelet lysate and laminin to enhance the neurotrophic effect of human adipose-derived stem cells
- Exploiting Caenorhabditis elegans to discover human gut microbiotamediated intervention strategies in protein conformational diseases
- How useful are biomarkers for the diagnosis of Alzheimer’s disease and especially for its therapy?
- N-methyl-D-aspartate receptor functions altered by neuronal PTP1B activation in Alzheimer’s disease and schizophrenia models
- The integrative role of G proteincoupled receptor heterocomplexes in Parkinson’s disease
- Brain-derived cell-free DNA
- Aminopeptidase A and dipeptidyl peptidase 4: a pathogenic duo in Alzheimer’s disease?
- Ubiquitin homeostasis disruption,a common cause of proteostasis collapse in amyotrophic lateral sclerosis?
- Transcranial electrical stimulation in neurological disease
- Novel insights into the pathogenesis of tendon injury:mechanotransduction and neuroplasticity
- Zinc finger protein ZPR1: promising survival motor neuron proteindependent modifier for the rescue of spinal muscular atrophy
- Complement activation kindles the transition of acute posttraumatic brain injury to a chronic inflammatory disease
- Degeneration of retinal ganglion cells in hypoxic responses: hypoxiainducible factor inhibition, a new therapeutic insight
Brain Injury and Neural Regeneration
- Bradykinin postconditioning protects rat hippocampal neurons after restoration of spontaneous circulation following cardiac arrest via activation of the AMPK/mTOR signaling pathway
- The delivery of miR-21a-5p by extracellular vesicles induces microglial polarization via the STAT3 pathway following hypoxia-ischemia in neonatal mice
- Temporal alterations in pericytes at the acute phase of ischemia/reperfusion in the mouse brain
- Cranial irradiation impairs intrinsic excitability and synaptic plasticity of hippocampal CA1 pyramidal neurons with implications for cognitive function
- A neurovascular unit-on-a-chip: culture and differentiation of human neural stem cells in a three-dimensional microfluidic environment
Spinal Cord Injury and Neural Regeneration
- A comparative analysis of differentially expressed genes in rostral and caudal regions after spinal cord injury in rats
- The functional properties of synapses made by regenerated axons across spinal cord lesion sites in lamprey
- Expression and regulatory network of long noncoding RNA in rats after spinal cord hemisection injury
Neurodegenerative Diseases and Neural Regeneration
- Anodal transcranial direct current stimulation alleviates cognitive impairment in an APP/PS1 model of Alzheimer’s disease in the preclinical stage
- Toxicities of amyloid-beta and tau protein are reciprocally enhanced in the Drosophila model
- SARM1 participates in axonal degeneration and mitochondrial dysfunction in prion disease
- DNA vaccines targeting amyloid-β oligomer ameliorate cognitive deficits of aged APP/PS1/tau triple-transgenic mouse models of Alzheimer’s disease